As a result, it was reasonable the system of inhibition of important WT1 isoform-mediated transcriptional activation of Bcl-xL and Bcl-2 by Ex4a(+)WT1 isoform may be level of competition with big WT1 isoform for interacting using the transcriptional co-activators besides WT1 by itself, resulting from the abrogation of major WT1 isoform-mediated transcriptional exercise in leukemia cells. Having sa